Burn Management all Phases

PRE-HOSPITAL BURN MANAGEMENT

1. Small thermal (<10% TBSA) - cool, clean, damp towel

2. Large thermal

ABCs

- airway patency, soot around nares, singed hair

- breathing adequacy

- pulse presence, regularity

3. Chemical

remove chemical (brush off)

lavage; blot dry

remove clothes

clean sheet

4. Electrical

remove from source

ABCs

C-spine precautions

exit/entry site

distal pulses

assess for injury (d/t falls, muscle contractions)

dry, clean sheet

EMERGENT PHASE

Phase during which immediate problems are addressed/resolved

Fluid loss/edema formation until fluid mobilization/diuresis

Pathophysiologic Problems

Fluid and electrolyte shifts:

Burn injury > increased blood flow > release of vasoactive substances > increased capillary permeability > water, Na, plasma protein shifts to interstitial and surrounding tissues > decreased intravascular volume > hypovolemic shock and its S&S.

Insensible losses (evaporation) - up to 400 ml/hour

RBCs:

- hemolyze d/t direct injury and circulating factor

- Hct elevated d/t hemoconcentration (r/t fluid loss)

- Net anemia (evident after fluid replacement)

Electrolytes

- K+ released from injured cells > hyperkalemia

- Na > interstitial spaces

RX: fluid resuscitation restores capillary normal permeability > stpos fluid losses and edema > interstitial fluid returns to vascular space > diuresis

Inflammation and Healing

leukocytes/monocytes accumulate

wound repair 6-12 hours after injury

Immunologic Changes: High infection risk d/t:

loss of protective skin barrier

reduced levels immunoglobulins

reduced #/function WBCs

Clinical Manifestations (Emergent Phase)

pain & hypovolemia > S/S shock

blisters (partial thickness burns)

edema

adynamic ileus (r/t trauma and K+ shift)

shivering (heat loss, anxiety, pain)

altered mental status (r/t hypoxia -- smoke inhalation; medications)

Complications (Emergent Phase)

CVS

shock, arrythmias

impaired circulation to extremities r/t circumferential burns/edema > ischemia, necrosis, gangrene

RX: Escharotomy (incision through eschar to restore

circulation

Increased blood viscosity > sludging

Respiratory

Upper airway burn > obstruction > asphyxia

Inhalation injury: alveolar damage d/t inhalation of chemical fumes or smoke > edema interferes with oxygenation

Renal

Hypovolemia >renal obstruction > ARF

Myoglobin (from muscle breakdown) & hemoglobin (RBC breakdown) can occlude renal tubules > ARF. Prevent with fluid replacement and diuretics

Management: Emergent Phase

ABC: intubate before respiratory failure develops; extubate when edema resolves. Escharotomy may be performed

Circulation:

- IV access

- Fluid replacement if > 20% TBSA with crystalloids

- Parkland Formula

- Second 24 hours: Colloids

Goal:

- U/O 30-50 ml/hr

- SBP > 90-100 mm Hg (via arterial line)

- HR < 100/mon

- Alert and oriented

WOUND CARE

Stabilize first

Cleanse and debride

Wound RX methods: Open or closed

Wound closure via graft

- heterograft

- homograft

- autograft

- cultured epithelial graft

PHARMACOLOGICAL MANAGEMENT

Analgesics/sedatives (IV)

Tetanus

Antimicrobials: Topical

- Flamazine, Acticoat, Gentamycin/Garamycin topically,

Bactigras, Porcine

NUTRITIONAL MANAGEMENT

Nasogastric tube for paralytic ileus until bowel sounds

Clear fluids > progress (high cal/protein)

Hypermetabolic: needs 5000 cal/day

ACUTE PHASE

Time from fluid mobilization/diuresis until burns are completely covered or healed (Duration: weeks/months)

Edema resolves

Burned areas more evident:

- partial thickness: pink-red, waxy/shiny, painful

- full thickness: dry, waxy white-dark brown, no sensation

Bowel sounds return

Healing begins; necrotic tissue sloughs

Clinical Manifestations:

Eschar separates: debride and graft

Partial thickness: self-healing

Hypo/hypernatremia

Hyper/hypokalemia

Complications (Acute Phase)

Infection

Cardio-pulmonary (as for Emergent Phase)

Neuro: extreme disorientation

Musculoskeletal: high risk for contractures***

GI:

- diarrhea (feedings, antibiotics)

- constipation (narcotics, immobility)

- ileus (sepsis)

- ulcers (stress): prevent with H2 histamine blockers

Endocrine: stress diabetes

Management Acute Phase:

Fluid therapy

Wound care

- Debridement (enzymatic, surgical)

- Excision and grafting: sheet graft, mesh graft

- Cultured epithelial autograft

- Artificial skin

Pain Management

- Partial thickness: excruciating

- Full thickness: no pain, except at margins (partial

thickness)

- Narcotic, psychotrophics

- Adjuncts: relaxation, imagery, etc.

Nutrition: high caloric needs; formula; supplements(po, TPN)

Emotional support (pain, disfigurement, body image, functional capacity, rehab)

REHABILITATIVE PHASE

Begins when wounds covered with skinor are healed; self-care begins!

Healed skin: pink and flat > then raised and hyperemic

New tissue: needs ROM to prevent contractures

Pressure garments to keep scars flat

Complications:

Contractures: keep body parts extended

EMOTIONAL SUPPORT!!!!!