Supplemental Table 1. List of References for the Interactions/Regulations Depicted in Figure 2

Supplemental Table 1. List of references for the interactions/regulations depicted in Figure 2.

Interaction/regulation number / Reference for interaction/regulation
1 / Bonora, M., et al., Tumor necrosis factor-alpha impairs oligodendroglial differentiation through a mitochondria-dependent process. Cell Death Differ, 2014. 21(8): p. 1198-208.
2 / Zou, Y., et al., Oligodendrocyte precursor cell-intrinsic effect of Rheb1 controls differentiation and mediates mTORC1-dependent myelination in brain. J Neurosci, 2014. 34(47): p. 15764-78.
3 / Cao, K., et al., AMPK activation prevents prenatal stress-induced cognitive impairment: Modulation of mitochondrial content and oxidative stress. Free Radical Biology and Medicine, 2014. 75(0): p. 156-166.
4 / St-Pierre, J., et al., Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators. Cell, 2006. 127(2): p. 397-408.
5 / Chen, Y., et al., The NFKB1 polymorphism (rs4648068) is associated with the cell proliferation and motility in gastric cancer. BMC Gastroenterol, 2015. 15(1): p. 21.
6 / Glauert, H.P., et al., The Role of NF-kappaB in PPARalpha-Mediated Hepatocarcinogenesis. PPAR Res, 2008. 2008: p. 286249.
7 / Paintlia, A.S., et al., Modulation of Rho-Rock signaling pathway protects oligodendrocytes against cytokine toxicity via PPAR-alpha-dependent mechanism. Glia, 2013. 61(9): p. 1500-17.
8 / Li, Z., et al., Chemically Diverse Toxicants Converge on Fyn and c-Cbl to Disrupt Precursor Cell Function. PLoS Biology, 2007. 5(2): p. e35.
9 / Miyake, S., et al., The tyrosine kinase regulator Cbl enhances the ubiquitination and degradation of the platelet-derived growth factor receptor alpha. Proc Natl Acad Sci U S A, 1998. 95(14): p. 7927-32.
10 / Heldin, C.H., A. Ostman, and L. Ronnstrand, Signal transduction via platelet-derived growth factor receptors. Biochim Biophys Acta, 1998. 1378(1): p. F79-113.
11 / Guardiola-Diaz, H.M., A. Ishii, and R. Bansal, Erk1/2 MAPK and mTOR signaling sequentially regulates progression through distinct stages of oligodendrocyte differentiation. Glia, 2012. 60(3): p. 476-86.
12 / Liu, X.M., et al., Activation of AMPK stimulates heme oxygenase-1 gene expression and human endothelial cell survival. Am J Physiol Heart Circ Physiol, 2011. 300(1): p. H84-93.
13 / Zhu, Z., et al., Prenatal stress causes gender-dependent neuronal loss and oxidative stress in rat hippocampus. J Neurosci Res, 2004. 78(6): p. 837-44.
14 / Maldonado-Cedillo, B.G., et al., Prenatal malnutrition and lead intake produce increased brain lipid peroxidation levels in newborn rats. Nutr Neurosci, 2015.
15 / Dibble, C.C. and L.C. Cantley, Regulation of mTORC1 by PI3K signaling. Trends Cell Biol, 2015. 25(9): p. 545-55.
16 / Gorentla, B.K. and X.P. Zhong, T cell Receptor Signal Transduction in T lymphocytes. J Clin Cell Immunol, 2012. 2012(Suppl 12): p. 5.
17 / Goncalves, R.L., et al., Sites of superoxide and hydrogen peroxide production by muscle mitochondria assessed ex vivo under conditions mimicking rest and exercise. J Biol Chem, 2015. 290(1): p. 209-27.
18 / Rego, A.C. and C.R. Oliveira, Mitochondrial dysfunction and reactive oxygen species in excitotoxicity and apoptosis: implications for the pathogenesis of neurodegenerative diseases. Neurochem Res, 2003. 28(10): p. 1563-74.
19 / Dixon, S.J. and B.R. Stockwell, The role of iron and reactive oxygen species in cell death. Nat Chem Biol, 2014. 10(1): p. 9-17.
20 / Yu, L. and S.J. Yang, AMP-activated protein kinase mediates activity-dependent regulation of peroxisome proliferator-activated receptor gamma coactivator-1alpha and nuclear respiratory factor 1 expression in rat visual cortical neurons. Neuroscience, 2010. 169(1): p. 23-38.
21 / Valko, M., et al., Free radicals and antioxidants in normal physiological functions and human disease. The International Journal of Biochemistry & Cell Biology, 2007. 39(1): p. 44-84.
22 / Shen, Q., et al., The role of pro-inflammatory factors in mediating the effects on the fetus of prenatal undernutrition: implications for schizophrenia. Schizophrenia research, 2008. 99(1-3): p. 48-55
23 / Diz-Chaves, Y., et al., Prenatal stress increases the expression of proinflammatory cytokines and exacerbates the inflammatory response to LPS in the hippocampal formation of adult male mice. Brain, behavior, and immunity, 2013. 28: p.196-206.