Jui-Yen Huang
2700 E 10th street, Apt 4, Bloomington, IN, 47408
| (919)597-8098
EDUCATION
2004/8-2010/8 Ph.D., Institute of Basic Medical Science, College of Medicine, National Cheng-Kung University, Tainan, Taiwan.
2003/9-2004/8 M.S., Department of Physiology, College of Medicine, National Cheng-Kung University, Tainan, Taiwan.
1999/9-2003/6 B.S., Nursing, College of Medicine, National Cheng-Kung University, Tainan, Taiwan.
RESEARCH EXPERIENCE
2015/4-present Postdoctoral Fellow in Department of Psychological and Brain Science, Indiana University Bloomington, Bloomington, IN, United States of America
2011/7-2015/3 Postdoctoral Associate in Department of Pediatric Neurology, Baylor College of Medicine, Houston, TX, United States of America
2010/8-2011/6 Postdoctoral Researcher in Department of Physiology, National Cheng-Kung University, Tainan, Taiwan
2003/8-2010/8 Graduate Research Assistant in Department of Physiology, National Cheng-Kung University, Tainan, Taiwan
1999/9-2003/6 Undergraduate Research Assistant in Department of Physiology, National Cheng-Kung University, Tainan, Taiwan
TEACHING EXPERIENCE
2004/8-2010/8 Teaching Assistant in Department of Physiology, National Cheng-Kung University, Tainan, Taiwan. Coordinate and teach the Laboratory of hearing physiology for MD students, undergraduate students
2003/9-2004/8 Teaching Assistant in Department of Physiology, National Cheng-Kung University, Tainan, Taiwan. Coordinate and teach the Laboratory of renal function test for MD students, undergraduate students
HONOR AND AWARDS
2015 Travel Award,Keystone Symposia Scholarship
2011 Poster Award, 19th Symposium on Recent Advances in Cellular and Molecular Biology
2010 Excellent Research Award in Institute of Basic Medical Sciences, National Cheng Kung University
2009 Poster Award, 17th Symposium on Recent Advances in Cellular and Molecular Biology
RESEARCH INTERESTS
Neurodegenerative disease
Redox biology
Cortical brain circuitry formation
Neurobiology
SOCIETY MEMBERSHIP
2008-Present Society for Neuroscience
2009-Present The Chinesae Society of Cell and Molecular Biology
PUBLICATION
1. Chuang JI, Huang JY, Tsai SJ, Sun HS, Yang SH, Chuang PC, Huang BM and Ching CH (2015) FGF9-induced changes in cellular redox status and HO-1 upregulation are FGFR-dependent and proceed through both ERK and AKT to induce CREB and Nrf2 activation. Free Radic Biol Med 89, 274-286.
2. Tsou YH,Shih CT,Ching CH,Huang JY,Jen CJ,Yu L,Kuo YM,Wu FS andChuang JI (2015) Treadmill exercise activates Nrf2 antioxidant system to protect the nigrostriatal dopaminergic neurons from MPP+ toxicity. Exp Neurol, 263, 50-62
3. Jew CP, Wu CS, Sun H, Zhu J, Huang JY, Yu D, Justice NJ and Lu HC (2013) mGluR5 Ablation in Cortical Glutamatergic Neurons Increases Novelty-Induced Locomotion. PLoS One, 8, e70415.
4. Huang JY, and Chuang JI (2010). Fibroblast growth factor 9 upregulates heme oxygenase-1 and gamma-glutamylcysteine synthetase expression to protect neurons from 1-methyl-4-phenylpyridinium toxicity. Free Radic Biol Med 49, 1099-1108.
5. Huang JY, Hong YT, and Chuang JI (2009). Fibroblast growth factor 9 prevents MPP+-induced death of dopaminergic neurons and is involved in melatonin neuroprotection in vivo and in vitro. J Neurochem 109, 1400-1412.
6. Lin CH, Huang JY, Ching CH, and Chuang JI (2008). Melatonin reduces the neuronal loss, downregulation of dopamine transporter, and upregulation of D2 receptor in rotenone-induced parkinsonian rats. J Pineal Res 44, 205-213.
CONFERENCE PODIUM TALKS/ POSTER PRESENTATION (Selected)
ABSTRACTS
1. Huang JY, Lu HC. Abnormal mGluR5 activity upregulate NGF/TrKA signaling in cortical glutamatergic neurons leading to aberrant dendritic morphogenesis. 45th Annual Meeting of the Society for Neuroscience, Chicago, IL, USA, 2015. (Nanosymposium / Oral Presentation)
2. Huang JY, Jason Michael Graf, Lu HC. FGFR3 hyperfunction causes aberrant development in cortex and hippocampus. 10th Gill Symposium, Bloomington, IN, USA, 2015.
3. Huang JY, Bakhshizadehmahmoudi D, Lu HC. Glutamate neurotransmission regulates FGF10 expression to modulate dendritic morphogenesis in developing cortical neurons. 10th Gill Symposium, Bloomington, IN, USA, 2015.
4. Huang JY, Ballester-Rosado CJ. Lu HC. Abnormal mGluR5 activity upregulate NGF/TrKA signaling in cortical glutamatergic neurons leading to aberrant dendritic morphogenesis. The Keystone Symposium on Pathways of Neurodevelopmental Disorders, Tahoe City, CA, USA, 2015.
5. Huang JY, Ballester-Rosado CJ. Lu HC. Defective neuronal morphogenesis in the absence of mGluR5 is due to enhanced NGF signaling through an increase in calcium-permeable AMPA receptors. 27th Barrel Meeting, Baltimore, MD, USA, 2014.
6. Huang JY, Ballester-Rosado CJ. Lu HC. Defective neuronal morphogenesis in the absence of mGluR5 is due to enhanced NGF signaling through an increase in calcium-permeable AMPA receptors. 9th Gill Symposium, Bloomington, IN, USA, 2014.
7. Huang JY, Tsai SJ. Chuang JI. FGF9 activates ERK1/2 and AKT / CREB and Nrf2 signaling pathway to induce ROS reduction and HO-1 upregulation. 19th Symposium on Recent Advances in Cellular and Molecular Biology, Pingtung, Taiwan, 2011.
8. Huang JY, Chuang JI. Neuroprotection of fibroblast growth factor 9 against 1-methyl-4-phenylpyridinium intoxication is attributed to the upregulation of antioxidant enzymes. 17th Symposium on Recent Advances in Cellular and Molecular Biology, Pingtung, Taiwan, 2009.
9. Huang JY, Chuang JI. FGF9 protects neurons from 1-methyl-4-phenylpyridinium intoxication by upregulating gene expression of antioxidant enzymes. 38th Annual Meeting of the Society for Neuroscience, Washington DC, USA, 2008.
10. Huang JY, Chuang JI. Melatonin rescues nigral dopaminergic neuronal death by reducing MPP+-induced FGF9 down-regulation. 17th Biennial Meeting of the International Society for Developmental Neuroscience. 2008, Asilomar, USA.
SCHOLARLY ACTIVITIES
1. Local organizing committee, Neuronal Coding 2009, Tainan, Taiwan.